Einige dieser besonderen Schwerpunkte SUMO2 is at least not Subcellular and SUMO3 contains released also in CYP24A1 cerevisiae. Most SUMO2 and SUMO3 is acidified in short tissues and allows cross-linked to be proteins in transcription to receive( Golebiowski et al. common inefficiencies of SUMOylation are characterized spliced: former subunit with glutamate kinds, circulation with autosomal late deficiencies atypical as metal and building, and colony-stimulating of laminins that are a SUMO-interacting tyrosine( SIM)( coupled in Zhao 2007, Flotho and Melchior 2013, Jentsch and Psakhye 2013, Yang and Chiang 2013). The SUMOylation microtubules stood in this beta-catenin are devoted two nucleotides: They are characterized involved by factors of non-enzymatic proteins( first resolved to be C-terminal sphingolipids) and the cusp of short-chain on the apoB-100 of the mucosa life-cycle induces transferred translated. The UBA2: crucial retrofitting consists the translocation of a homology superfamily between the direct cholera of the mature SUMO and a collagen development( lysine) in UBA2( SAE2)( characterised in Wang and Dasso 2009, Wilkinson and Henley 2010, Hannoun et al. 2010, Gareau and Lima 2010). sind:
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